Insulin-like growth factor-I, inflammatory proteins, and fibrosis in subjects with nonalcoholic fatty liver disease
- Autori: Hribal, M.; Procopio, T.; Petta, S.; Sciacqua, A.; Grimaudo, S.; Pipitone, R.; Perticone, F.; Sesti, G.
- Anno di pubblicazione: 2013
- Tipologia: Articolo in rivista (Articolo in rivista)
- Parole Chiave: Fibrosis; Humans; Aged; Fatty Liver; Insulin-Like Growth Factor I; Inflammation; Cross-Sectional Studies; Liver; Middle Aged; Biological Markers; Cell Line; Female; Male
- OA Link: http://hdl.handle.net/10447/73250
Inflammation may have a pathogenic role in the progression of nonalcoholic fatty liver disease (NAFLD); by contrast, the role of anti-inflammatory molecules has not been addressed. Low circulating levels of the anti-inflammatory molecule IGF-I have been described in subjects with NAFLD.